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The drugs mimic a hormone your gut produces after you eat, telling your brain you're full, slowing digestion, and making you eat less

genetic deletion or small-molecule inhibition of ALKBH5 reduces these suppressive infiltrates and improves anti-PD-1 efficacy in multiple syngeneic models, directly tying an m 6 A eraser to the composition of tumor-infiltrating immune cells

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Overall, the role of autophagy in establishing the follicular pool and promoting follicular survival highlights its potential as a novel therapeutic target for addressing DOR in vulnerable populations, such as those exposed to chemotherapy, environmental toxins, or genetic disorders (Figure 3B)

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